AIDS & Empire


Dr. Nancy Banks – AIDS, Opium, Diamonds & Empire

Dr Nancy Turner-Banks

Dr. Nancy Banks is a graduate of Hunter College and Harvard Medical School. She completed her internship and residency in general surgery and obstetrics and gynecology. Today she is an activist, speaker and author of AIDS, Opium, Diamonds and Empire. Nancy will talk about the conspiratorial side of the medical industry and the elite cabal behind it who are skilled in the art of war. She also talks about how the HIV/AIDS crisis developed from the consequences of global financial decisions made at the highest levels as the result of the Bretton Woods Agreement following WWII and the doping of America following Vietnam. We’ll also discuss the Rockefeller Institute, eugenics and the art of propaganda and deception. Later, we cover biological stressors and how to stay healthy from the constant physical onslaught.

Radio 3Fourteen:
Listen as Dr. Nancy Banks discusses her book, “AIDS, Opium, Diamonds and Empire”

Dr. Nancy Banks - Download MP3



HIV Does Not Cause AIDS


Bauer, H. H. (2010, April-June). HIV Does Not Cause AIDS. Edge Science #3, pp. 6-9.

“What everyone knows” is sometimes wrong. When it comes to science, including medical science, history might even suggest that what everyone knows at any given time turns out later to have been wrong to some degree: scientific understanding has progressed, after all, and it has often progressed by overturning earlier theories.

But even as it’s widely recognized that science has progressed, it’s usually forgotten that this very progress has often meant superseding or rejecting earlier ideas. And the notion that a contemporary consensus might be wrong seems unbelievable to most people.

STOP hiv

So the claim that HIV doesn’t cause AIDS, when everyone knows that it does, is treated by the media, the public, and mainstream science as not worth attending to. And yet the proof that HIV cannot be the cause of AIDS is at hand in the technical literature, as well as in dozens of books aimed mainly at a general audience.

To consider that proof, it’s necessary not only to specify that evidence but also to provide some assurance that good alternative explanations are available for what AIDS is and what HIV is. So those questions will be answered after outlining the reasons why the HIV=AIDS theory is wrong.

The Evidence that HIV Doesn’t Cause AIDS…

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Bauer-H-H – 2010-April-June – HIV Does Not Cause AIDS – Edge Science #3 – pp 6-9




By Stefan Lanka
Continuum (UK) April/May 1995

Stefan Lanka

An error can never become true however many times you repeat it. The truth can never be wrong, even if no one ever hears about it.
~Mahatma Gandhi

For the past 10 years or so it has been the accepted wisdom that the Human Immuno-deficiency Virus, HIV, causes AIDS. It supposedly occurs in many body fluids, and its transmission especially in semen and blood to a new host, triggers a slow but inexorable progression to AIDS and ultimately death. To infect another cell, HIV must at some stage in its life cycle exist as a separate and identifiable entity.

What has been ignored and kept from public awareness is, that there has never been a workable HIV test and that the definition of ‘positive’ has always changed according to the views of different organisations dealing with it, changed also according to the kind of tests used and changed from laboratory to laboratory performing the tests:

“.. Its techniques have not been standardised, and the magnitude and consequences of interlaboratory variations have not been measured. Its results require interpretation, and the criteria for this interpretation vary not only from laboratory to laboratory but also from month to month …” (1)

The dispute over who discovered HIV (2) was a distraction from the question of whether the virus actually exists at all.

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Looking Back on the Oxidative Stress Theory of Aids


Looking Back On the Oxidative Stress Theory of AIDS

Eleni Papadopulos-Eleopulos
Department of Medical Physics, Royal Perth Hospital, Perth, Western Australia

Looking back on the Oxidative Stress theory of Aids
Eleni Papadopulos-Eleopulos
Department of Medical Physics, Royal Perth Hospital, Perth, Western Australia
Continuum vol 5, no 5, 1999

The whole purpose of a scientific theory is to explain the mechanism behind observations and make predictions. If a theory cannot explain the observations for which it was put forward, or if its predictions are not fulfilled, then it should be abandoned. In this regard, despite the lapse of 18 years, there is still no proof as to the cause(s) of AIDS. Of course, there are theories but the biggest obstacle in overcoming the problem of AIDS, and proving its cause, is that one of these theories, the HIV theory, has been uncritically accepted since 1984. However, of all the theories, the HIV is the least likely.”

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Looking Back on the Oxidative Stress Theory of AIDS – Eleni Papadopulos-Eleopulos – Continuum v5n5 1999



Vol 4, No 3 September/October 1996
Eleni Papadopulos-Eleopulos(1) Valendar F. Turner(2) John M. Papadimitriou(3) David Causer(1)
(1) Department of Medical Physics, (2) Department of Emergency Medicine, Royal Perth Hospital, Perth, Western Australia; (3) Department of Pathology, University of Western Australia.

Listening to both sides of a story will convince you that there is more to a story than both sides” ~ Frank Tyger

Has the Isolation & Purification of 'HIV' Really Been Achieved?

The definite existence of any virus, including a retrovirus, can be proven only by isolating it. For nearly half a century retroviruses have been isolated by banding in density gradients. It is accepted that the procedures incorporated into this method, which is by no means perfect, have not been followed by the researchers who claim isolation of the human immunodeficiency virus, HIV-1. Nonetheless, it is said that at present, there is ample evidence that HIV has been isolated and shown to be a unique exogenous retrovirus.”

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The Isolation of HIV – Has It Really Been Achieved – The Case Against – E Papadopulous-Eleopulous – Continuum v4n3 Sep-Oct 1996



Roberto A. Giraldo, MD
Continuum, vol 5, no 5, 1999

Medical researcher Roberto A. Giraldo, MD is obtaining astonishing results

Roberto Giraldo MD

Physician, specialist in internal medicine, infectious and tropical diseases. Member of the Boards of Directors of The Group for the Scientific Reappraisal of the HIV-AIDS Hypothesis and Health Education AIDS Liaison (HEAL). Independent AIDS Researcher. Author of the book ‘AIDS and Stressors,’ New York City. <

“For the last 6 years I have been working at a laboratory of clinical immunology in one of the most prestigious University Hospitals in the City of New York. Here I have had the opportunity to personally run and get to know in detail the current tests used for the diagnosis of HIV status, namely, the ELISA, Western blot and Viral Load tests”


1. Diluting the serum for the ELISA test

The ELISA test is a test for antibodies against what is supposed to be the Human Immunodeficiency Virus or HIV. To run this test, an individual’s serum has to be diluted to a ratio of 1:400 with a special specimen diluent. According to the test kit manufacturer this diluent contains 0.1% triton X-100, Bovine and Goat Sera (minimum concentration of 5%) and Human T-Lymphocyte Lysate (minimum titer 1:7500). Preservative: 0.1% Sodium Azide (1)

This extraordinarily high dilution of the person’s serum [400 times] took me by surprise. Most serologic tests that look for the presence of antibodies against germs uses neat serum [undiluted]. For example, the tests that look for antibodies to hepatitis A and B viruses, rubella virus, syphilis, hystoplasma and cryptococus, to mention a few of them, use straight serum [undiluted]. However, to try to prevent false positive reactions some serologic tests use diluted serum; for example this is the case with tests that look for antibodies to measles, varicelia and mumps viruses which use a dilution of 1:16, to cytomegalovirus [CMV] 1:20 and to Epstein-Barr Virus [EBV] 1:10.

The obvious questions are: What makes HIV so unique that the test serum needs to be diluted 400 times? And what would happen if the individual’s serum is not diluted?

2. Testing the ELISA test without diluting the serum

To answer these questions I ran an experiment in a medical laboratory in Yorktown Heights, New York. I ran it using the same test kit reagents that are usually used to run the ELISA test in most clinical laboratories worldwide (1).

I first took samples of blood that, at 1:400 dilution, tested negative for antibodies to HIV. I then ran the exact same serum samples through the test again, but this time without diluting them. Tested straight, they all came positive.

Since that time I have run about 100 specimens and have always gotten the same result. I even ran my own blood which, at 1:400, reacts negative. At 1:1 [undiluted] it reacted positive. I should mention that with the exception of my own blood, the patient samples all came from doctors who requested HIV tests. It is therefore likely that most of the blood samples that I tested belonged to individuals at risk for AIDS.

According to Abbott Laboratories, the absorbance value [yellow color intensity]

develops in proportion to the amount of antibodies to HIV-1 which is bound to the bead (1).

What I noticed is that the absorbance values of the specimens that tested negative when diluted [1: 400], but positive when undiluted [1:1], had lower values than the samples that, diluted, react positive on both the ELISA and Western blot tests. This would probably mean that the blood that is negative when diluted but positive when undiluted has a lower level of antibodies than the diluted blood that is doubly positive and, therefore, may probably test negative on the Western blot test. However, I have not had the opportunity to check this hypothesis.

The graphic below illustrates how blood that reacts negative for HIV at 1:400 ratio always turn positive when run at 1:1 [undiluted].

FireShot Screen Capture #001 - 'HIV & AIDS - Everybody reacts positive on the ELISA test for HIV' - htm

Column (a) shows 10 specimens reacting negative at 1:400 dilution.
Column (b) shows the same specimens reacting positive at 1:1 dilution.

It is important to note that the Western blot antibody test for “HIV” also needs serum to [be] diluted. Although it too has an unusually high dilution, here the individual serum is only diluted at a ratio of 1:50 (2). I have not yet had the opportunity to run this test with undiluted [1:1] specimens.

3. Discussion

The following are three possible explanations for why undiluted specimens of blood always react positive at the ELISA test:

3. 1. Everybody has HIV antibodies.

It is accepted worldwide that the ELISA test for HIV detects antibodies against what is known as the Human Immunodeficiency Virus (3-6). And the pharmaceutical company that commercializes the ELISA kits states that

Abbott HIVAB HIV-1 EIA is an in vitro qualitative
Enzyme Immunoassay for the Detection of Antibody
to Human Immunodeficiency Virus Type 1 (HIV-1) in
Human Serum and Plasma (1).

Since all undiluted blood specimens react positive on the ELISA test, a test that supposedly tests for antibodies to HIV, the results presented here suggest that every single human being has HIV antibodies. And this suggests that everybody has been exposed to HIV antigens.

This would mean that all of us have been exposed to the virus that is believed to be the cause of AIDS. The people that react positive even at a dilution of 1:400, would be the ones that have had the highest level of exposure to HIV antigens. The rest of the people – the ones that only react positive with undiluted serum [1:1] – would have had a lower level of exposure to HIV.

3.2. Everybody has different levels of HIV infection.

It is also believed worldwide that a person that reacts positive for antibodies against HIV has not only been exposed to but is infected with a deadly virus that causes immunodeficiency (3-6). Therefore, the positive reactions of all undiluted sera would mean that everybody, or at least all the blood samples that I have tested, including my own, infected with this “deadly” virus. The ones that react positive at a ratio of 1:400 would simply have a higher level of “deadly” infection than the “deadly” infection had by the ones that react positive only with undiluted serum.

3.3. The test is not specific for HIV

The results presented here could also mean that the tests used for detecting antibodies to HIV are not specific for HIV, as has been explained previously (7-14). In this case, there would be reasons other than HIV infection, past or present, to explain why a person reacts positive to it. The test also reacts positive in the absence of HIV (7-14).

The scientific literature has documented more than 70 different reasons for getting a positive reaction other than past or present infection with HIV (7,10,11,14,15). All these conditions have in common a history of polyantigenic stimulations (15,16).

Even Abbott Laboratories is well aware of the specificity problems with the ELISA test. This is why they state:

EIA testing alone cannot be used to diagnose AIDS,
even if the recommended investigation of reactive
specimens suggests a high probability that the
antibody to HIV-1 is present


Although for all clinical and public health applications
of the EIA both the degree of risk for HIV-1
infection of the person studied and the degree of
reactivity of the serum may be of value in interpreting
the test, these correlations are imperfect.
Therefore, in most settings it is appropriate to investigate
repeatably reactive specimens by additional
more specific or supplemental tests (1).

Interestingly, there are countries like Great Britain where the diagnosis of HIV status is based on the ELISA test alone. No Western blot or any other test is needed there.

The only proper way for establishing the sensitivity and specificity of a given test is with a gold standard. However, since HIV has never been isolated as an independent purified viral entity (17-19), there cannot be a gold standard for HIV. The sensitivity and specificity of the antibody tests for HIV have instead been defined based on the assumption that HIV is the cause of AIDS. In this way,

The Abbott studies show that: Sensitivity based on
an assumed 100% prevalence of HIV-1 antibody in
AIDS patients is estimated to be 100% (144 patients


Specificity based on an assumed zero prevalence of
HIV-1 in random donors is estimated to be 99.90/o
(4777 random donors tested) (1).

At present there is no recognized standard for
establishing the presence and absence of HIV-1
antibody in human blood. Therefore sensitivity was
computed based on the clinical diagnosis of AIDS
and specificity based on random donors (1).

[Emphasis is mine].

Since there is no scientific evidence that the ELISA test is specific for HIV antibodies, a reactive ELISA test at any concentration of the serum would mean presence of nonspecific or polyspecific antibodies (20). These antibodies could be present in all blood samples. They are most likely a result of the stress response, having no relation to any retrovirus, let alone HIV (21,22). In this case, a reactive test could be a measure of the degree of one’s exposure to stressor or oxidizing agents (15,16).

The inevitable conclusion is that all positive reactions for antibodies to HIV are simply false positives. If nobody is positive for HIV, then people who react positive on the ELISA test do so due to something other than HIV.

4. Proposal to find out the real meaning of the “HIV
antibody” tests.

To uncover the meaning of these tests I propose a simple experiment: Take blood from three groups of people and run the tests highly diluted, undiluted and at a wide spectrum of dilutions in between. The first group would be a group of healthy people of many age groups; the second group would be a group of people from the conventional AIDS “risk groups”; the third group would be a group of people with clinical conditions both related and unrelated to AIDS. All groups would be subjected to both the ELISA and Western blot tests.

Additionally, all blood samples could be subjected to the “the viral load test for HIV”.

The results of such an experiment could determine whether these test measurements bear any relationship to an individual’s level of exposure to stressor or oxidizing agents. If so, the tests could be salvaged as a measure of an individual’s level of intoxication.

Let us find the economic support necessary to run this experiment. In the mean time, since people are reacting positive on tests that are not specific for HIV, let’s please stop labeling them as “HIV positive”.

Read Full PDF (Includes References):
Everbody Reacts Positive On The Elisa Test For HIV – Giraldo – Continuum 1999 v5n5

REVIEW: Demystified . . . Human endogenous retroviruses


Screen Capture www_ncbi_nlm_nih_gov_pmc_articles_PMC1187282_pdf_mp56000011


J Clin Pathol; Mol Pathol; 2003;56:11-18
P N Nelson, P R Carnegie, J Martin, H Davari Ejtehadi, P Hooley, D Roden,
S Rowland-Jones, P Warren, J Astley, P G Murray

Human endogenous retroviruses (HERVs) are a family of viruses within our genome with similarities to present day exogenous retroviruses. HERVs have been inherited by successive generations and it is possible that some have conferred biological benefits. However, several HERVs have been implicated in certain cancers and autoimmune diseases. This article demystifies these retroviruses by providing an insight into HERVs, their means of classification, and a synopsis of HERVs implicated in cancer and autoimmunity. Furthermore, the biological roles of HERVs are explored.”

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The viruses in all of us: Characteristics and biological significance of human endogenous retrovirus sequences



Proceedings of the National Academy of Sciences

The viruses in all of us:
Characteristics & biological significance of human endogenous retrovirus sequences
(reverse transcriptase/retroelements/human teratocarcinoma-derived virus)

Roswitha Lӧwer, Johannes Lӧwer, and Reinhard Kurth
Paul-Ehrlich-lnstitut, Paul Ehrlich Strasse 51-59, D-63225 Langen, Germany
Communicated by Maurice R. Hilleman, Merck Research Laboratories, West Point, PA, December 15, 1995

“Human endogenous retroviruses (HERVs) are very likely footprints of ancient germ-cell infections. HERV sequences encompass about 1% of the human genome [*8-10% - See NOTE Below]. HERVs have retained the potential of other retroelements to retrotranspose and thus to change genomic structure and function. The genomes of almost all HERV families are highly defective. Recent progress has allowed the identification of the biologically most active family, HTDV/HERV-K, which codes for viral proteins and particles and is highly expressed in germ-cell tumors. The demonstrable and potential roles of HTDV/HERV-K as well as of other human elements in disease and in maintaining genome plasticity are illustrated.”

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[*EDITOR'S NOTE: More recent published findings from the human genome project have revealed to the scientific community that approximately 8-10% of the human genome consists of endogenous retroviral sequences, and overall, the virus-like components of the human genome amount to almost 50% of our DNA.]

Cell Membrane Vesicles Are a Major Contaminant of Gradient-Enriched Human Immunodeficiency Virus Type-1 Preparations


VIROLOGY 230, 125–133 (1997)

Cell Membrane Vesicles Are a Major Contaminant of Gradient-Enriched Human Immunodeficiency Virus Type-1 Preparations

"Purified HIV-1 preparations are contaminated by cellular vesicles. Purified vesicles from H9 cells (a)..".Gluschankof, P. et al. Cell membrane vesicles are a major contaminant of gradient-enriched human immunodeficiency virus type-1

“Purified HIV-1 preparations are contaminated by cellular vesicles. Purified vesicles from H9 cells..”.



During preliminary experiments to establish the proportion of virus-coded p24 protein to virus membrane-associated HLA-DR in gradient-enriched HIV-1 preparations, we became aware of a large variability between experiments. In order to determine whether HLA-DR-containing cellular material was contaminating the virus preparations, we carried out enrichment by gradient centrifugation of clarified supernatants from noninfected cells and tested this material for HLA-DR content. We found that, independently of the cell type used, gradient enrichment resulted in the isolation of large quantities of HLA-DR-containing material which banded at a density overlapping that of infectious HIV. Electron microscopy of gradient-enriched preparations from supernatants of virus-infected cells revealed an excess of vesicles with a size range of about 50–500 nm, as opposed to a minor population of virus particles of about 100 nm. Electron micrographs of infected cells showed polarized vesiculation of the cell membrane, and virus budding was frequently colocalized with nonviral membrane vesiculation. Analysis of the cellular molecules present in the fractions containing virus or exclusively cellular material demonstrated that virus and cellular vesicles share several cellular antigens, with the exception of CD43 and CD63, found mainly at the virus surface, and HLA-DQ, which was found only in the cellular vesicles.”

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Cell Membrane Vesicles Are a Major Contaminant of Gradient-Enriched Human Immunodeficiency Virus Type-1 Preparations

Microvesicles Are a Source of Contaminating Cellular Proteins Found in Purified HIV-1 Preparations


VIROLOGY 230, 134–144 (1997)

Microvesicles Are a Source of Contaminating Cellular Proteins Found in Purified HIV-1 Preparations

virol bess
Identification and quantitation of cellular proteins associated with HIV-1 particles are complicated by the presence of nonvirion-associated cellular proteins that copurify with virions. Many cellular proteins are associated with nonviral particles that bud from the surface of cells called microvesicles. Microvesicles band in sucrose gradients in a range of densities that includes the same density as retroviruses.”

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Human Genome Bears Virus Related to HIV (1999)


Proceedings of the National Academy of Sciences
November 09, 1999
Human Genome Bears a Virus Related to HIV-1
Howard Hughes Medical Institute


Article Highlights:

Buried within the genetic blueprint of every human is a snippet of DNA that resembles a gene sequence from the human immunodeficiency virus (HIV). Humans have been carrying this unwanted genetic baggage around for more than 30 million years, according to researchers from the Howard Hughes Medical Institute (HHMI) at Duke University.”

We’re all walking around with a little bit of an HIV-like sequence in our genes,” said Bryan R. Cullen, an HHMI investigator at Duke University.

According to Cullen and his colleagues, “an ancient family of viruses, known as HERV-K (for Human Endogenous RetroVirus K), took up permanent residence in the genetic material of monkeys. The viruses then traveled with their simian and pre-human hosts as these species moved along the evolutionary path that led to Homo sapiens.” Cullen’s group published its findings in the November 9, 1999, issue of the Proceedings of the National Academy of Sciences.

“…some viruses insert their DNA into the host’s genome and direct the host’s cellular machinery to make the proteins needed to assemble more viruses. If this gene insertion takes place in a cell that will become an egg or a sperm, the host’s offspring will have a copy of the virus in every single cell.” “Once it’s in there, it doesn’t get out,” Cullen said.

Because of these viral gene insertion events, genetic material from inactive viruses accounts for roughly 3 percent of the human genome.” [*8-10% - See Note Below]. . . “but there doesn’t seem to be a harmful effect from the activity of these genes,” Cullen said.

[*EDITOR'S NOTE: More recent published findings from the human genome project have revealed to the scientific community that approximately 8-10% of the human genome consists of endogenous retroviral sequences, and overall, the virus-like components of the human genome amount to almost 50% of our DNA.]

The consistent presence of Human Endogenous Retroviruses (HERVs) within the human genome continues to offer up intriguing insights into the evolution of both humans and viruses, including so-called “HIV.”

Howard Hughes Medical Institute.

Related Article:
Novel human endogenous sequences related to human immunodeficiency virus type 1
Endogenous retrovirus-related sequences exist within the normal genomic DNA of all eukaryotes, and these endogenous sequences have been shown to be important to the nature and biology of related exogenous retroviruses and may also play a role in cellular functions. …. Herein we describe the first report of the presence of nucleotide sequences related to HIV-1 in human, chimpanzee, and rhesus monkey DNAs from normal uninfected individuals.”
Journal of Virology 1992 April; 66(4): 2170–2179.

AIDS since 1984: No evidence for a new, viral epidemic – not even in Africa



AIDS since 1984: No Evidence
Italian Journal of Anatomy & Embryology
IJAE, Vol. 116, No. 2: 73-92, 2011
Peter H. Duesberg, Daniele Mandrioli1, Amanda McCormack1, Joshua M. Nicholson, David Rasnick, Christian Fiala, Claus Koehnlein, Henry H. Bauer and Marco Ruggiero

Since the discoveries of a putative AIDS virus in 1984 and of millions of asymptomatic carriers in subsequent years, no general AIDS epidemic has occurred by 2011. In 2008, however, it has been proposed that between 2000 and 2005 the new AIDS virus, now called HIV, had killed 1.8 million South Africans at a steady rate of 300,000 per year and that anti-HIV drugs could have saved 330,000 of those. Here we investigate these claims in view of the paradoxes that HIV would cause a general epidemic in Africa but not in other continents, and a steady rather than a classical bell-shaped epidemic like all other new pathogenic viruses. Surprisingly, we found that South Africa attributed only about 10,000 deaths per year to HIV between 2000 and 2005 and that the South African population had increased by 3 million between 2000 and 2005 at a steady rate of 500,000 per year. This gain was part of a monotonic growth trajectory spanning from 29 million in 1980 to 49 million in 2008. During the same time Uganda increased from 12 to 31 million, and Sub-Saharan Africa as a whole doubled from 400 to 800 million, despite high prevalence HIV. We deduce from this demographic evidence that HIV is not a new killer virus. Based on a review of the known toxicities of antiretroviral drugs we like to draw the attention of scientists, who work in basic and clinical medical fields, including embryologists, to the need of rethinking the risk-and-benefit balance of antiretroviral drugs for pregnant women, newborn babies and all others who carry antibodies against HIV.

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HIV Tests Are Not HIV Tests


HIV Tests Are Not HIV Tests
Journal of American Physicians & Surgeons Vol 15 No 1 Spring 2010
Henry H. Bauer, Ph.D.


Tests for human immunodeficiency virus (HIV) do not detect HIV; they respond “positive” to a wide range of physiological conditions. The seminal papers from Gallo’s laboratory did not demonstrate HIV to be the cause of AIDS. The patent based on those papers did not demonstrate that the proposed HIV tests, which are actually for HIV antibodies, are specific for HIV antibodies. From the original announcement that “HIV is the probable cause of AIDS” to “HIV antibodies demonstrate active infection by HIV” was an unwarranted progression that came almost subliminally. HIV test kits were approved only for blood screening and do not claim to diagnose infection. There is no gold standard for an HIV test; the existing tests are at most adjuncts to clinical diagnosis of actual infection by HIV.

Consequences of misapplication of “HIV” tests include subjecting healthy individuals to iatrogenic harm through life-long intake of highly toxic medication.

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Iatrogenic Harm Following “HIV” Testing


Iatrogenic Harm Following “HIV” Testing
Journal of American Physicians & Surgeons Vol 15 No 2 Summer 2010
Henry H. Bauer, Ph.D.


A large proportion of asymptomatic human immunodeficiency virus (HIV) “positive” individuals—50% or more—are likely never to progress to illness if left untreated. That follows from official estimates of numbers of undiagnosed “HIV positives,” numbers of known HIV/AIDS cases, numbers of AIDS deaths over the years, and from the high frequency of false positive tests that result from screening low-risk populations.

Nonetheless, antiretroviral treatment (ART) is, increasingly, being initiated purely on the basis of laboratory tests for HIV and CD4 cell counts, in the absence of symptoms. Perhaps half or more of HIV positive patients are needlessly taking drugs with highly debilitating side effects. At highest risk of iatrogenic harm are pregnant women, Africans, and persons of recent African ancestry.

The rationale for population-wide screening for human immunodeficiency virus (HIV) is timely initiation of treatment to prevent progression to symptomatic immune deficiency and early death, and also to prevent transmission, especially vertical transmission from mother to infant. Optimal decisions depend on characteristics of the test, the illness, and the treatment.

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Faculty of Mathematical, Physical and Natural Sciences
Degree in Biological Sciences
Department of Pathology and Experimental Oncology


Thesis of:
Chiara Matteuzzi

Supervisor: Prof. Stefania Pacini
Correlator: Prof. Marco Ruggiero

Academic Year: 2009-2010










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Human Endogenous Retroviruses and AIDS Research: Confusion, Consensus, or Science?


Human Endogenous Retroviruses & AIDS Research
Journal of American Physicians & Surgeons Vol 15 No 3 Fall 2010
Etienne de Harven, M. D.


Human Endogenous Retroviruses (HERVs) are confounding factors in HIV/AIDS research that cannot be ignored. Evidence suggests that “viral load” may actually be measuring retroviral nucleoside sequences associated with HERVs. HERVs also provide a valid explanation for the presence of retroviruses recognizable by electron microscopy (EM) in the original 1983 publication from the Institut Pasteur, and may account for claims of innumerable “mutations” of the putative HIV pathogen. The interference of HERVs in AIDS research brings into question the subject of study in so-called “AIDS Research,” and the very existence of an exogenous HIV pathogen itself.

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Reliability of PCR to Detect Genetic Sequences from HIV


AIDS - Knowledge and Dogma_ Congress, July 16_17 2010, Vienna, AustriaFrom the 2010 Alternative AIDS Conference in Vienna a must-read on polymerase chain reaction (PCR) and endogenous retroviral sequences:

Reliability of PCR to Detect Genetic Sequences from HIV
Juan Manuel Morillo Velázquez, PhD.

Juan Manuel Morillo Velázquez, PhD

Juan Manuel Morillo Velázquez, PhD


Polymerase chain reaction (PCR) is a lab technique used for detection of genetic sequences derived from microbial or human genes, so it can be used in microbiological and genetic diagnosis. Its applications in HIV/AIDS has been increasing in the last 15 years, in viral load testing to measure the number of copies of HIV RNA in human plasma, and for detection of HIV DNA (proviral DNA) within human cells. However, there are several studies reporting the low specificity of the method. One of the reasons could be the high genetic diversity of HIV, but other reason could be the presence of endogenous retroviral sequences in human samples. An approach to test the reliability those sequences usually employed in commercial tests, and to study the hypothesis that genetic sequences considered as HIV could be endogenous is presented.

Read Full PDF: Reliability of PCR to Detect Genetic Sequences from HIV

The Case Against ‘HIV’ – Dr. Henry Bauer

Dr. Henry Bauer’s excellent and extensive summary of the evidence against the ‘HIV=AIDS’ theory (Version 1.01, October 12, 2013):

Henry H. Bauer, PhD.

Henry H. Bauer, PhD.

“That HIV causes AIDS has been the officially sanctioned view for about 3 decades, believed almost universally but questioned openly by thousands of people, some of whom are expert in relevant sciences.  These dissidents point out that a comprehensive reading of the mainstream literature together with analysis of mainstream data demonstrates conclusively that HIV is neither a necessary nor a sufficient cause of AIDS.” … READ FULL PDF

Official Obituary For Tommy David “The Duke” Morrison

Tommy "The Duke" Morrison

Tommy “The Duke” Morrison

Tommy David Morrison “The Duke”, age 44, professional boxer, passed away, after suffering cardiac arrest, on Sunday, September 1, 2013. His death was not due to HIV as some sources have reported. Official pathology lab results confirmed there was no HIV present in his blood or body. Tommy was born January 2, 1969 in Gravette, Arkansas to Tim and Diana (Harrison) Morrison. He married Trisha Harding on May 7, 2011 in Sevierville, TN. In addition to his wife and his parents, Tommy is survived by his many fans; sons, Trey, McKenzie and Tristin; stepsons, Paul, Stephen and Justin; brother, Timmy; sister, Tonya; his aunt, Joy along with other aunts and uncles from both sides of the family, two grandchildren and other family members. Celebrations of his life are being held by his fans all over the world. Condolences may be sent to God Bless “The Duke”

Interview with Trisha Morrison about Tommy Morrison’s death
Robert Scott Bell Show • Robert Scott Bell interviews Trisha Morrison to talk about Tommy Morrison’s life and death in the context of HIV, starting with the (since vanished) positive HIV test in 1996. [LISTEN HERE] 

Viral Maize? GMO Corn Contained Alleged Key Protein Found on the Surface of “HIV”

12 April 2002

Edible HIV vaccine breakthrough

Maize genetically modified to contain a key protein found on the surface of the monkey form of HIV has been created by US company ProdiGene. This development brings an edible, more effective, HIV vaccine for people a step closer, says the US National Institutes of Health.”HIV-Ribbon_GMO_Corn

The protein – SIV gp120 – is the simian form of a protein that will be used in an HIV vaccine trial on people in Thailand later in 2002. Researchers will inject a canarypox virus modified to contain HIV genes and then a booster shot of HIV gp120. Both the trial and ProdiGene’s SIV maize research are being funded by the NIH.”


Fungus Found in Corn, Wheat, Rice & Nuts Silently Worsening “AIDS” Epidemic

A new study suggests that a type of fungus coating much of the stored corn, wheat, rice and nuts in developing countries may be quietly worsening the AIDS epidemic.” 


The study recruited 314 “HIV-positive people who were not yet on antiretroviral therapy” and divided patients into four groups based on their level of aflatoxin exposure and found that “those in the highest exposure group were 2.6 times more likely to have a high HIV viral load than those in the lowest exposure group.”

Of course, according to conventional “HIV” theory, the researchers are under the erroneous assumption that “Higher viral load translates into higher rates of HIV transmission and the potential for earlier progression to the opportunistic infections of AIDS.”


HIV NO MEDS STUDY – Texas A&M University Department of Health & Kinesiology


Decisions to stop (or never start) drug treatments for HIV infection: A pilot study of long-term nonprogressors (LTNPs)

Texas A&M University
“Anomalies”. That is how mainstream science and health care view persons who have tested positive for HIV infection, have refused to submit to mainstream treatments, yet have survived for a decade or longer without developing AIDS.

If LTNPs – also known as long-term survivors, long-term healthy seropositives, elite controllers, or elite suppressors – have captured the attention of geneticists, virologists and immunologists, they have, nonetheless, gone unnoticed by clinicians, policy makers, and health educators. Non-progressors remain, for all practical purposes, “anomalies”; mysterious cases for which science cannot easily account.

Specifically, very little is known about the lived experiences of HIV+ persons who refuse mainstream anti-retroviral therapies and remain AIDS-free for many years. Some of the questions we don’t have good answers for, include:

  • What shapes the decision not to start, or discontinue the highly active anti-retroviral therapies (HAART)?
  • What shapes the decisions to stand against a dominant culture of pharmacological preventive therapy for HIV infection? What does it take to go against the mainstream? How much stress does taking a stance against drug treatments, involve?
  • Do LTNPs who refuse HAART therapy believe the drug regimen could harm, instead of improve their health?
  • Do people from certain ethnic, or cultural groups, have specific reasons (unique to their group or their culture) for not starting or stopping HIV drug treatments?
  • Do men and women decide never to start or to stop drug treatments, for different reasons?

To begin answering these questions is the main objective of this study. And in order to answer these questions, we need to listen to the stories of people who have experienced exactly what we’re talking about, here: no drug treatment for HIV infection.

The Principal Investigator in this study is Dr. Pat Goodson. She is a health educator, and a professor in the Division of Health Education (Department of Health & Kinesiology). She has been at Texas A&M University for 11 years. One of her areas of research is the sexual health of adults and adolescents.

Pat is seeking participants for this study. If you, or someone you know, might be interested in participating, and would like to be interviewed, please check to see if you meet the criteria for participating in the study. We can only interview people who:

  • were diagnosed HIV positive at least 8 years ago
  • have not been diagnosed with AIDS (ever)
  • are not taking medication for HIV at the moment
  • are 18 years old or older

If interested, please contact Pat Goodson (or her research assistant) to set up an interview by PHONE or SKYPE:

Pat Goodson at (979) 845-1756 or

Check our more about our research on YouTube.